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Betty and Elyn represent good examples of the two types of myopathy that can result from elevated levels of steroid administration. As can be seen in Figure 1, a strong rise in the level of testosterone is noted with acute and subacute treatment, and this rise appears to be associated with a decline in the ability of the muscle fibers to contract quickly and efficiently. In addition, an increase in glycolytic ATP synthesis and the increased oxygen consumption leading to the hypertrophy induced by the treatment of myopathy have been noted. These differences between acute and subacute treatment may be due to a reduction in the number of total myopathic fibers, the type and number of myotonic dystrophy that is induced by acute treatment, and the extent to which steroid administration reduces myotonic dystrophy. Figure 1 Changes in muscle fibre length associated with chronic treatment with estradiol and/or testosterone in young men. It was previously hypothesized that an increased myopathy with acute steroid exposure is an adaptive response to a relatively limited amount of time (1 to 3 days) of exposure to testosterone. However, in the present series, there was no change in the average fibre length compared to the same values at baseline. This means that it is unlikely that an adaptive response existed that allowed a greater number of fibres to contract rapidly and efficiently in response to treatment. Interestingly, the change in fibre length was associated with the degree of myopathy, as the higher the degree of myopathy, the more lengthened the average fibre length. Moreover, during acute treatment, this myopathy was associated with an increased concentration of steroid administration, which was consistent with the increased muscle concentrations of testosterone in the acute treatment group. It was recently postulated that a decrease in the myopathy could occur due to an increase in the concentration of the steroid itself. Since the myopathy was not associated with any change in fibre length, it is possible that this effect may only be seen when steroid administration is administered in chronically increased hormone concentrations (10 to 20 microg of testosterone/kg). However, a decrease in fibre length is not expected in subjects undergoing frequent and high-intensity exercise, and thus there have been no reports of this phenomenon occurring. Furthermore, it should be noted that both treatment groups received a very large number of training sessions that they exercised regularly (6 weeks). This could be an indication of the increased muscular activity that is normally stimulated during low-intensity resistance training. The above results indicate that subacute steroid administration at low doses causes reductions in the myopathy in young men, and that this effect may be adaptive Related Article:
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